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Sedentary lifestyle claims more than 4 million direct victims each year
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According toarecent study, lack ofphysical activity is themain risk factor forpremature mortality fortheentire world population.
This paper highlights thepercentage ofdeaths that can be attributed tosedentary lifestyles worldwide. Analarming conclusion is that asedentary lifestyle is themain risk factor formany non-communicable diseases. These include cardiovascular disease, coronary artery disease, stroke, hypertension, andtype 2 diabetes. There are also cancers (bladder, stomach, kidney, breast, colon, andesophagus), depression, anddementia.
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Astudy published intheBritish Medical Journal ofSports Medicine onMarch 29, 2021, estimates that physical inactivity is responsible for7.2% ofall-cause deaths each year. This represents about 4 million deaths out ofanaverage of56.9 million peryear. TheAmerican andCanadian researchers behind this work obtained information from several databases indifferent states. Inaddition, they tried toget theresults closest toreality byconsidering pathologies whose causal relationship with asedentary lifestyle is well known. Inother words, we are talking about aserious level ofevidence. According totheresults, indeveloping countries, most deaths (inabsolute numbers) occur due tothelack ofphysical activity ofapart oftheir population. Ontheother hand, developed countries are more likely tosuffer from theeffects ofasedentary lifestyle, asthey are subject toamuch higher percentage ofdeaths. Thestudy's authors strongly believe that thepublic health burden associated with physical inactivity is aglobal problem that will require international cooperation tomobilize change andachieve these public health goals. Forresearchers, change should be focused onachieving public health goals. This means increased investment inthere-opening ofsports facilities during thepandemic. It also means making them available toasmany people aspossible inthelong run.
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Scientists have found anew method offighting antibiotic-resistant bacteria
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Stress often causes bacteria toform biofilms. Stress can manifest asaphysical barrier, ultraviolet light, oratoxic substance such asantibiotics. Theformation ofthese biofilms takes from several hours todays, andthey can be ofdifferent shapes, sizes, colors, andtextures depending onthetype ofbacteria. Being inabiofilm state protects them from harmful substances intheenvironment biofilms have aunique outer wall with different physical andchemical properties than their individual cells. They can coordinate metabolism, slow down their growth, andeven form animpenetrable barrier ofwrinkles andcreases. This is one oftheways they achieve high antibiotic resistance. Researchers from theUnited Kingdom recently studied thetransition oftheHay Bacillus bacterium from afree-moving swarm toabiofilm asadefense mechanism andpublished what they did tocombat its antibiotic-resistant properties ineLife.

Todetermine if their test strain was behaving like theothers, they first ran stress tests onthem. They tested thebacteria's response tothephysical barrier, ultraviolet light, andantibiotics. Theaddition ofaphysical barrier led tothetransition ofbacteria from one layer toamultilayer layer, followed byanincrease incell density andtheformation ofmultilayer islands near thebarrier. Later, wrinkles formed ontheislands near thebarrier intheplace where they began toappear initially.
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When they applied ultraviolet light totheswarm, they again observed adrop incell speed andanincrease indensity. Andafter thescientists added alarge dose oftheantibiotic kanamycin, thebacterial cells formed abiofilm. Theresearchers then developed astrategy tocombat this bacterial biofilm. They added kanamycin totheenvironment ofanew batch ofswarming bacterial cells andwatched asthebiofilm began toform. They then re-injected theantibiotic atamuch higher dose than thefirst, just before thebiofilm formation was complete. Asaresult, thepartially formed biofilm was destroyed andbacterial cells died. This shows that antibiotic-resistant bacteria lose their resistance toantibiotics when they undergo aphase transition, right before going into thebiofilm, where they will become much more resistant. Thus, with thecorrect administration ofantibiotics, thebacteria can be attacked intheir most vulnerable state anddestroyed. Theresearchers believe that similar transitions from swarm tobiofilm occur inother bacterial species. Their research may pave theway forfinding more effective ways tocontrol clinically relevant bacteria. Forexample, Salmonella enterica, which spreads into thebloodstream andis transmitted through contaminated food. OrPseudomonas aeruginosa with multiple drug resistance, which after surgery causes infections intheblood, lungs (pneumonia) andother parts ofthebody andspreads inhospitals.
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